With great honor, Dr. Raj Ratan, executive director of Burke Neurological Institute, has been invited to serve as a member of the Neural Oxidative Metabolism and Death Study Section, Center for Scientific Review for the term beginning July 01, 2018 and ending June 30, 2024.
Thiamine (vitamin B1) dependent processes are reduced in Alzheimer’s disease. Thiamine deficiency mimics many aspects of Alzheimer’s disease including the reduced glucose metabolism and exaggeration of the plaque and tangle pathology.
The Board of Trustees, Faculty and Staff of the Burke Medical Research Institute, an affiliate of Weill Cornell Medicine mourn the loss of our founder, friend and colleague.
Thiamine (vitamin B1) deficiency (TD) produces a mild, chronic impairment of oxidative metabolism that models the diminished metabolism and reduced activities of the thiamine-dependent mitochondrial enzymes that occur in brain in several common age-related neurodegenerative disorders.
On March 2nd, a manuscript from the Burke Medical Research Institute describing a novel drug for improving outcomes following brain hemorrhage was published in Science’s sister journal, Science Translational Medicine.
