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Role of CD36 in Functional Recovery in Chronic Stroke

September 30, 2015 to August 31, 2021
GRANT: 
Federal
Funding Status: 
Completed Project
Funding Agency: 
National Institutes of Health (NIH)
Funding Institute: 
National Institute of Neurological Disorders and Stroke (NINDS)
Grant Number: 
R01NS095359
Published Grant: 
NIH RePORT

Goals

This proposal addresses the mechanism(s) by which peripheral immunity regulated by CD36 contributes to functional recovery in chronic stroke.

Related Research Project

Cho Lab
RESEARCH PROJECT: 
In Progress
The Role of CD36 in Stroke-Induced Inflammation and Brain Injury

Despite a well-documented role of CD36 in the pathogenesis of conditions such as atherosclerosis, inflammation and lipid metabolism, its role in cerebral ischemic injury had not been recognized at the time I initiated the st

Investigators

Sunghee Cho, Ph.D.
Lab Director
Stroke Neuroimmunology Laboratory
Professor
Weill Cornell Medicine
Role: 
Principal Investigator

Associated

Research Labs: 
Cho Lab

Conditions & Recovery

Stroke
Stroke is the leading cause of disability in the U.S.

Research Methods

Molecular Studies