Mitochondrial aconitase is a transglutaminase 2 substrate: transglutamination is a probable mechanism contributing to high-molecular-weight aggregates of aconitase and loss of aconitase activity in Huntington disease brain.

PUBLICATION:

Journal Article

Authors:

Kim SY, Marekov L, Bubber P, Browne SE, Stavrovskaya I, Lee J, Steinert PM, Blass JP, Beal MF, Gibson GE, Cooper AJ.

Year Published:

2005

Publisher:

Neurochem Res. 2005 Oct;30(10):1245-55.

Identifiers:

PMID:16341586

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New York-Presbyterian Hospital

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Laboratory for Mitochondrial Biology and Metabolic Dysfunction in Neurodegeneration

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